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While our study confirmed previous observations that spinal cord injury induces degeneration of spinal motoneurons with loss of dendritic branches, axonal terminals and presynaptic boutons [27], [30], [33]–[35], it also demonstrated that the degenerative processes rostral and caudal to the injury epicenter progressed asymmetrically with significantly less motoneurons remaining above the lesion site

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The major detoxification pathway of 4HNE involves GSTs 16